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  • HumanApolipoproteinA-IVReducesSecretionofProinflammatoryCytokinesandAtheroscleroticEffectsofaChronicInfectionMimickedbyLipopolysaccharide

    FromUnitéd’ExpressiondesGènesEucaryotes(D.R.,M.A.O.,A.-L.G.-O.,J.P.,M.M.Z.),InstitutPasteur,Paris,France;UnitédeGénétiqueMycobactérienne(E.B.),InstitutPasteur,Paris,France;U545Inserm(G.C.),InstitutPasteurandFacultédePharmacie,UniversitéLilleII,Lille,France;andUnitéd’Immuno-Hématologiee...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2004年第24卷第4期
  • InheritedApolipoproteinA-VDeficiencyinSevereHypertriglyceridemia

    FromtheDepartmentofBiomedicalSciences(C.P.O.,P.T.,S.C.),UniversityofModenaandReggioEmilia;DepartmentofInternalMedicine(L.P.,A.B.,S.B.),UniversityofGenova;DepartmentofPediatrics(G.L.V.,M.P.S.),UniversityofCatania;NationalInstituteofHealth(A.C.),Rome;DepartmentofPharmacologicalSciences(A.C.),Universit...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2005年第25卷第2期
  • PreventionofCoronaryHyperreactivityinPreatherogenicMenopausalRhesusMonkeysbyTransdermalProgesterone

    FromDimeraIncorporated(R.K.H.,R.G.M.)andOregonHealthandScienceUniversity(D.P.,B.U.,M.K.A.,K.A.B.,D.R.I.),Portland,Ore;UniversityofSouthernCalifornia(F.Z.S.),LosAngeles,Calif;St.George’sHospitalMedicalSchool(J.C.K.),London,UK;andRheinConsultingLaboratories(F.J.N.),Portland,Ore.ABSTRACTObjective—T...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2004年第24卷第5期
  • IL-20IsExpressedinAtherosclerosisPlaquesandPromotesAtherosclerosisinApolipoproteinE-DeficientMice

    【摘要】Objective-Atherosclerosisisachronicinflammatorydiseasewithimmunecellinfiltration.Variouscytokinesandchemokineshavebeencharacterizedaspro-orantiatherogenicfactors.Interleukin-20(IL-20)belongstotheIL-10familyandisaproinflammatorycytokineinvolvedinthepathogenesisofpsoriasis.However,theassoc...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2006年第26卷第9期
  • PhospholipidTransferProteinAugmentsApoptosisinTHP-1-DerivedMacrophagesInducedbyLipolyzedHypertriglyceridemicPlasma

    【摘要】Objective-Lipolysisoftriglyceride-richlipoproteins(TGRLPs)generatesphospholipid-richsurfaceremnantsandinducescytotoxiceffectsinadjacentvascularcells.WehypothesizedthatbyintegratingsurfaceremnantsintoHDL,phospholipidtransferprotein(PLTP)alleviatescytotoxicity.MethodsandResults-Totestthish...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2007年第27卷第4期
  • AscorbateSupplementReducesOxidativeStressinDyslipidemicPatientsUndergoingApheresis

    FromDepartmentsofMedicalResearch(C.T.-C,H.-W.C.,),InternalMedicine(W.-T.C.,T.-D.W.,Y.-T.L.),andPathology(S.-M.H.),NationalTaiwanUniversityCollegeofMedicineandNationalTaiwanUniversityHospital,Taipei;FormosanBloodPurificationFoundation(S.-Y.L.),Taipei;DepartmentofHealth(T.-J.C.),Taichung;DepartmentofB...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2004年第24卷第6期
  • AtypicalGPI-AnchoredT-CadherinStimulatesAngiogenesisInVitroandInVivo

    【摘要】Objective-T-cadherin(T-cad)isanatypicalGPI-anchoredmemberofthecadherinsuperfamily.Invasculartissue,T-cadexpressionisincreasedduringatherosclerosis,restenosis,andtumorneovascularization.Invitro,overexpressionand/orhomophilicligationofT-cadonendothelialcells(ECs)facilitatesmigration,prolif...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2006年第26卷第10期
  • SelectiveCyclooxygenase-2InhibitionWithCelecoxibDecreasesAngiotensinII-InducedAbdominalAorticAneurysmFormationinMice

    【摘要】Objective-Inflammationplaysanintegralroleinthedevelopmentofabdominalaorticaneurysms(AAAs),andtheexpressionofcyclooxygenase(COX)-2isincreasedinaneurysmaltissuecomparedwithnormalaorta.Nonsteroidalanti-inflammatorydrugs,whichinhibittheactivityofCOX-1andCOX-2,decreaseAAAexpansioninhumansanda...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2006年第26卷第5期
  • EffectofP-SelectinonPhosphatidylserineExposureandSurface-DependentThrombinGenerationonMonocytes

    FromtheSectionofCardiology(I.D.C.,N.S.K.),ThrombosisResearchSection(I.D.C.,P.T.,J.A.L.,N.S.K.),DepartmentofPathology(P.T.),andDepartmentofMedicine(F.N.),BaylorCollegeofMedicine,Houston,Texas;andServeid’HemoterapiaiHemostasia(R.T.),HospitalClinic,Barcelona,Spain.CorrespondencetoNealS.Kleiman,MD,The...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2005年第25卷第5期
  • Glucosamine-InducedEndoplasmicReticulumStressPromotesApoB100Degradation

    FromtheDivisionofClinicalBiochemistry(W.Q.,R.K.-A.,S.M.,J.T.,K.A.),theDepartmentofLaboratoryMedicinePathobiology,HospitalforSickChildren,UniversityofToronto,Ontario,Canada;andtheDepartmentofPathologyandMolecularMedicine(R.C.A.),McMasterUniversity,Hamilton,Ontario,Canada.CorrespondencetoKhosrowAdeli,...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2005年第25卷第3期

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