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  • ReducedImmunoregulatoryCD31+TCellsintheBloodofAtheroscleroticMiceWithPlaqueThrombosis

    FromEMI0016(G.C.,A.A.H.Z.,J.S.,D.U.,M.L.,A.L.),FacultédeMédecineParis,France;U681(E.G.,J.K.-L.,A.-T.G.,A.N.),InstitutdesCordeliers,INSERM,Paris,France.CorrespondencetoGiuseppinaCaligiuri,MD,PhD,INSERMU681,InstitutdeCordeliers;15,ruedel’EcoledeMédecine,75006Paris,France.E-mailcaligiuri@necker.fr...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2005年第25卷第8期
  • DysregulationoftheUbiquitin-ProteasomeSysteminHumanCarotidAtherosclerosis

    【摘要】Objective-Theubiquitin-proteasomesystemistheprincipaldegradationrouteofintracellularandoxidizedproteins,thusregulatingmanycellularprocessesconceivablyimportantforatherosclerosis.Theaimofthisstudywastoevaluatetheactivityofubiquitin-proteasomesysteminhumancarotidarteryplaquesinrelationtoox...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2006年第26卷第9期
  • FarnesoidXReceptorLigandsInhibitVascularSmoothMuscleCellInflammationandMigration

    【摘要】Objective—ThefarnesoidXreceptor/bileacidreceptor(FXR;NR1H4)isaligand-activatedtranscriptionfactorthatregulatesbileacidandlipidhomeostasis,andishighlyexpressedinenterohepatictissue.FXRisalsoexpressedinvasculartissue.WehaveinvestigatedwhetherFXRregulatesinflammationandmigrationinvasc...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2007年第27卷第12期
  • 丝虫

    ...聚集在肺内的有效刺激不是那里氧压的绝对水平,而是肺动脉内静脉血和肺静脉内的动物血两者间的氧张力之差。当氧张力差在7.3kPa(55mmHg)或更高时,微丝蚴聚集于肺血管内;差异下降到接近5.9kPa(44mmHg)或更低时,微丝蚴则移行...

    词条生物学;寄生虫;人体寄生虫;线虫
  • 天然生物支架材料在软骨修复中的研究进展

    ...前已经有研究应用同种异体脱细胞材料修复膀胱、尿道、动脉和皮肤缺损[1~3],以及构建心脏瓣膜[4],结果比较满意。另外关于脱细胞软骨基质和小肠粘膜下基质(SIS)这2种天然的细胞外基质材料应用也有相关的研究报道[...

    参考资料医源资料库;在线期刊;中国矫形外科杂志;2006年第14卷第6期
  • 第八节 丝虫

    ...聚集在肺内的有效刺激不是那里氧压的绝对水平,而是肺动脉内静脉血和肺静脉内的动物血两者间的氧张力之差。当氧张力差在7.3kPa(55mmHg)或更高时,微丝蚴聚集于肺血管内;差异下降到接近5.9kPa(44mmHg)或更低时,微丝蚴则移行...

    参考资料医源资料库;医源图书馆;教材类;人体寄生虫学
  • MacrophageApolipoproteinEReducesAtherosclerosisandPreventsPrematureDeathinApolipoproteinEandScavengerReceptor-ClassBIDouble-KnockoutMice

    【摘要】Objective-Micenullforbothapolipoprotein(apo)Eandscavengerreceptor(SR)-BI(DKO)developseverehypercholesterolemia,occlusivecoronaryatherosclerosis,myocardialinfarction,andprematuredeath.ThecurrentstudyexaminestheabilityofmacrophageapoEtoimprovethedyslipidemia,reduceatherosclerosis,andrescue...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2006年第26卷第1期
  • DietaryFat–InducedAlterationsinAtherosclerosisAreAbolishedbyACAT2-DeficiencyinApoB100Only,LDLr–/–Mice

    【摘要】Objectives—Theenzymeacyl-coenzymeA(CoA):cholesterolO-acyltransferase2(ACAT2)intheliversynthesizescholesterylesters(CE)fromcholesterolandfattyacyl-CoA,whichgetincorporatedintoapoB-containinglipoproteinsthataresecretedintothebloodstream.Dietaryfattyacidcompositioninfluencestheamounta...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2007年第27卷第6期
  • RegulationofXanthineOxidoreductaseProteinExpressionbyHydrogenPeroxideandCalcium

    FromtheDivisionofCardiology(J.S.M.,A.S.,S.D.,D.G.H.),MolecularandSystemsPharmacologyProgram(J.S.M.,D.G.H.),EmoryUniversity,Atlanta,Ga;theAtlantaVeteransHospitalMedicalCenter,Atlanta,Ga;andtheDepartmentofMedicine(H.C.),UniversityofChicago,Ill.CorrespondencetoDavidG.Harrison,DivisionofCardiology,Emory...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2005年第25卷第8期
  • HumanLecithin:CholesterolAcyltransferaseDeficiency

    【摘要】Objectives-Lecithin:cholesterolacyltransferasedeficiency(LCAT-def)ischaracterizedbylowlevelsofhigh-densitylipoprotein(HDL)andlow-densitylipoprotein(LDL)andtheaccumulationoflipoprotein-X(LpX).DespitethelowHDL,atherosclerosisisuncommoninLCAT-def.ThedecreasedLDLwouldbeapossibleexplanationbu...

    参考资料医源资料库;在线期刊;动脉硬化血栓血管生物学杂志;2006年第26卷第6期

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