FromtheDepartmentofMolecularMedicineandMetabolism(T.S.,J.N.,Y.O.)andtheCenterofExcellenceProgramforFrontierResearchonMolecularDestructionandReconstitutionofToothandBone(Y.O.),MedicalResearchInstitute,TokyoMedicalandDentalUniversity,Tokyo,JapanCorrespondencetoYoshihiroOgawa,MD,Ph.D.,DepartmentofMolec...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2005年第25卷第10期【摘要】Objective-Endothelialdysfunctionpredictsmorbidityandmortalityinpatientsatcardiovascularrisk.Endothelialfunctionmaybedecisivelyinfluencedbythedegreeofendothelialcellapoptosis.MethodsandResults-Totestthishypothesisinhumans,endothelial-dependentvasodilatationwasinvasivelyassessedin50patient...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2006年第26卷第1期FromtheLipidSciencesResearchProgram(L.L.R.,R.L.),theDepartmentsofPathologyandBiochemistry,WakeForestUniversitySchoolofMedicine,Winston-Salem,NC;andtheCenterforMetabolismandEndocrinology(P.P.),DepartmentofMedicine,KarolinskaInstituteatKarolinskaUniversityHospital,Huddinge,Sweden.CorrespondencetoLawre...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2005年第25卷第6期【摘要】Objective-Theroleofthenuclearreceptorperoxisome-proliferatoractivatedreceptor(PPAR)-ß/inendothelialcellsremainsunclear.Interestingly,theselectivePPARß/ligandGW501516isinphaseIIclinicaltrialsfordyslipidemia.Here,usingGW501516,wehaveassessedtheinvolvementofPPARß/inendothe...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2007年第27卷第1期【摘要】Objective-Weexploredtherelationshipsbetweeninflammatoryproteins,occupation,andmaritalstatus,andtheirindependentassociationswithincidenceofcardiovasculardisease(CVD).MethodsandResults-Fiveinflammation-sensitiveproteins(ISPs)(fibrinogen,ceruloplasmin,haptoglobin,1-antitrypsin,orosomucoid)w...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2006年第26卷第3期【摘要】Background-BoththemetabolicsyndromeandanincreasedapolipoproteinB/AI(apoB/AI)ratioarepowerfulriskfactorsforcardiovascularevents.WehypothesizedthattheapoB/AIratiowell-characterizesthedyslipidemiaassociatedwithinsulinresistanceandthemetabolicsyndromeandinvestigatedthoserelationsandiftheapoB...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2006年第26卷第2期【摘要】Thisarticleexploresthephysiologyofsuperoxidegenerationbyendothelialnitricoxidesynthase(eNOS),theso-calleduncoupledstateoftheenzyme.ThefactthatthisalternativechemistryoftheeNOSenzymeisevolutionarystronglyconserved,suggeststhatitmayplayaphysiologicalrole.Itisproposedthatthisuncoupledstatem...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2006年第26卷第2期FromtheDepartmentofSurgery,CollegeofPhysiciansSurgeons,ColumbiaUniversity,NewYork,NY.SeriesEditor:RichardA.CohenATVBInFocusDiabeticVascularDisease:PathophysiologicalMechanismsintheDiabeticMilieuandTherapeuticImplicationsABSTRACTReceptorforAGE(RAGE)isamulti-ligandmemberoftheimmunoglobulinsuperfamilyo...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2004年第24卷第8期FromtheMedicalCollegeofGeorgia,Augusta,Ga.ABSTRACTAlthoughtheimportanceofproteintyrosinephosphorylationbytyrosinekinasesinmitogenicsignalingiswell-accepted,recentstudiesalsosuggestthattyrosinedephosphorylationbyproteintyrosinephosphatases(PTPases)playanequallyimportantrole.Forexample,bothangiotensin...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2004年第24卷第11期【摘要】Objective-Weassessedtheclinicalsignificanceofintima(IT),media(MT),andintima-media(IMT)thicknessofthecommoncarotidarteryusingB-modeultrasonographicimageprocessing.MethodsandResults-Onehundredseventyconsecutivepatientsunderwentcommoncarotidarteryscanningusinghigh-resolutionultrasonography....
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2006年第26卷第10期