FromtheWallenbergLaboratoryforCardiovascularResearch(E.K.R.,A.K.,C.U.,K.E.,O.W.,B.G.O.,L.M.H.);theResearchCenterforEndocrinologyMetabolism(P.-A.S.,L.M.S.C.),SahlgrenskaUniversityHospital,G?teborg,Sweden;andMolecularPharmacology(A.-C.J.-R,W.M.)andDMPK,BioanalyticalChemistry(G.I.H.)AstraZenecaRD,M?lnd...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2004年第24卷第11期FromtheDepartmentofObstetricsandGynaecology(Y.Y.,S.-K.L.);DepartmentofSurgery(J.Q.,R.M.E.O.);DepartmentofPathology(M.T.);NationalUniversityMedicalInstitute(P.C.),NationalUniversityofSingapore;andGenomeInstituteofSingapore(S.-K.L.),Singapore.Tobetterunderstandendothelialdifferentiation,aninvitrocellu...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2004年第24卷第4期【摘要】Objective-Weinvestigatedthemolecularmechanismofnicotine-acceleratedatherosclerosisinahyperlipidemiclow-densitylipoproteinreceptor-/-mousemodel.MethodsandResults-Low-densitylipoproteinreceptor-/-micereceivedtime-releasenicotineorplacebopelletsfor90days.Aorticlesionsizewas2.5timeslargerinn...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2006年第26卷第1期【摘要】Objective—Vascularendothelialgrowthfactor(VEGF),akeyangiogenicgrowthfactor,stimulatesangiogenesis.Lowlevelsofreactiveoxygenspecies(ROS)functionassignalingmoleculesforangiogenesis.WepostulatedthatlowconcentrationsofoxLDLmightinducelowlevelsofROSandinitiateangiogenesis.MethodsandResu...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2007年第27卷第11期【摘要】Objective-Thrombinactivatablefibrinolysisinhibitor(TAFI)attenuatesfibrinolysis.TheaimofthepresentstudywastoinvestigatethepossibleassociationbetweenTAFIandoverallischemicstrokeandischemicstrokesubtypes.MethodsandResults-TheSahlgrenskaAcademyStudyonIschemicStroke(SAHLSIS)comprises600cases(...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2007年第27卷第4期FromtheDepartmentofPharmacologicalSciences(L.S.,E.G.,P.G.,U.G.,E.N.,A.G.,B.C.,R.P.,E.T.),CentreforExcellenceonNeurodegenerativeDiseases,(L.S.,E.G.,R.P.,E.T.),ProteomicandProteinStructureStudyGroup(E.G.),UniversityofMilan;andMonzinoCardiologicCenterIRCCS(E.T.),Milan,Italy.CorrespondencetoLuigiSironi,...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2005年第25卷第3期【摘要】Background-WehypothesizedthatatherosclerosisinhibitsNO-mediatedendothelium-dependentdilationofcoronaryarteriolesthroughinteractionofox-LDLwithitsreceptor,LOX-1,throughtheproductionofO2ÿ-inendothelialcells.MethodsandResults-Weassessedtheroleofox-LDLinendothelialdysfunctioninamurinemo...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2007年第27卷第4期【摘要】Objective-ThisworkwasundertakentoinvestigatecomparativeeffectofAT1receptorblocker(ARB),3-hydroxy-3-methylglutaryl(HMG)coenzymeA(CoA)reductaseinhibitor(statin),andtheircombinationonvascularinjuryofsalt-sensitivehypertension.MethodsandResults-Salt-loadedDahlsalt-sensitivehypertensiverats(D...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2007年第27卷第3期FromtheDivisionofMolecularMedicine(S.B.S.,A.R.T.,C.L.W.),theDepartmentofMedicine,InstituteofHumanNutrition(S.B.S.,C.D.L.,R.L.L.,A.R.T.),andtheDivisionofMolecularGenetics(C.D.L.,R.L.L.),DepartmentofPediatrics,ColumbiaUniversity,NewYork;andtheLaboratoryofBiochemicalGeneticsandMetabolism(J.L.B.),TheRoc...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2005年第25卷第1期【摘要】Objective-Theproproteinconvertasesubtilisin/kexintype9(PCSK9)geneencodesaproproteinconvertasethatcausesdegradationofcellsurfacelow-densitylipoproteinreceptors(LDLRs).MutationsinthePCSK9genethatdisruptthenormalfunctionofPCSK9couldthereforeresultinincreasednumberofLDLRsandhypocholesterolem...
参考资料医源资料库；在线期刊；动脉硬化血栓血管生物学杂志；2006年第26卷第5期